Point mutation in AML1 disrupts subnuclear targeting, prevents myeloid differentiation, and effects a transformation-like phenotype

Diana Vradii, Sayyed K. Zaidi, Jane B. Lian, Andre J. Van Wijnen, Janet L. Stein, Gary S. Stein

Research output: Contribution to journal › Article › peer-review

37 Scopus citations

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Medicine & Life Sciences