V617F "JAKs" up myeloproliferative signal

Ayalew Tefferi

doi:10.1182/blood-2005-08-3352

V617F "JAKs" up myeloproliferative signal

Ayalew Tefferi

Hematology

Research output: Contribution to journal › Review article › peer-review

Abstract

JAK2^V617F, an ostensibly myeloid lineage-specific mutation, also appears to be myeloid disease-specific according to 2 studies in the current issue of Blood, which also features a third study that identifies altered gene expression in polycythemia vera as a surrogate for JAK-STAT hyperactivation.

Original language	English (US)
Pages (from-to)	3335-3336
Number of pages	2
Journal	Blood
Volume	106
Issue number	10
DOIs	https://doi.org/10.1182/blood-2005-08-3352
State	Published - Nov 15 2005

ASJC Scopus subject areas

Biochemistry
Immunology
Hematology
Cell Biology

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10.1182/blood-2005-08-3352

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title = "V617F {"}JAKs{"} up myeloproliferative signal",

abstract = "JAK2V617F, an ostensibly myeloid lineage-specific mutation, also appears to be myeloid disease-specific according to 2 studies in the current issue of Blood, which also features a third study that identifies altered gene expression in polycythemia vera as a surrogate for JAK-STAT hyperactivation.",

author = "Ayalew Tefferi",

year = "2005",

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doi = "10.1182/blood-2005-08-3352",

language = "English (US)",

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journal = "Blood",

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AU - Tefferi, Ayalew

PY - 2005/11/15

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AB - JAK2V617F, an ostensibly myeloid lineage-specific mutation, also appears to be myeloid disease-specific according to 2 studies in the current issue of Blood, which also features a third study that identifies altered gene expression in polycythemia vera as a surrogate for JAK-STAT hyperactivation.

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M3 - Review article

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SP - 3335

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JO - Blood

JF - Blood

IS - 10

ER -

V617F "JAKs" up myeloproliferative signal

Abstract

ASJC Scopus subject areas

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