Sudden cardiac death

Research output: Contribution to journalArticle

3 Citations (Scopus)

Abstract

Sudden cardiac death can occur after exposure to extreme stress and sometimes as a complication of acute neurologic disease. Excessive adrenergic stimulation of the heart is most likely the responsible mechanism for the majority of cases of sudden cardiac death. The neurocardiogenic injury induced by sympathetic overstimulation can affect the myocardium and the electrical conduction system, leading to heart failure and arrhythmias. The characteristic features of stress cardiomyopathy (also known as takotsubo cardiomyopathy or apical ballooning syndrome) can be diagnosed by echocardiography and cardiovascular magnetic resonance imaging. This chapter reviews the history, definition, pathophysiology, triggers, and clinical manifestations of neurocardiogenic injury. It also discusses specific neurologic conditions associated with sudden death: epilepsy (sudden unexplained death in epilepsy, SUDEP) and stroke.

Original languageEnglish (US)
Pages (from-to)19-24
Number of pages6
JournalHandbook of Clinical Neurology
Volume119
DOIs
StatePublished - Jan 1 2014

Fingerprint

Takotsubo Cardiomyopathy
Sudden Cardiac Death
Sudden Death
Epilepsy
Wounds and Injuries
Acute Disease
Nervous System Diseases
Adrenergic Agents
Nervous System
Echocardiography
Cardiac Arrhythmias
Myocardium
Heart Failure
History
Stroke
Magnetic Resonance Imaging

Keywords

  • Apical ballooning syndrome
  • Neurocardiogenic injury
  • Stress cardiomyopathy
  • Sudden death
  • SUDEP
  • Takotsubo

ASJC Scopus subject areas

  • Medicine(all)
  • Neurology
  • Clinical Neurology

Cite this

Sudden cardiac death. / Rabinstein, Alejandro.

In: Handbook of Clinical Neurology, Vol. 119, 01.01.2014, p. 19-24.

Research output: Contribution to journalArticle

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