Hypokalemia, aldosteronism, and renal cysts

A. B. Weder; P. M. Stewart; C. R.W. Edwards; Michel Azizi; Christiane Battaglia; Gilles Chatellier; Pierre François Plouin; Pierre Corvol; Vicente E. Torres; William F. Young; Kenneth P. Offord; Robert R. Hattery

doi:10.1056/NEJM199007123230212

Hypokalemia, aldosteronism, and renal cysts

A. B. Weder, P. M. Stewart, C. R.W. Edwards, Michel Azizi, Christiane Battaglia, Gilles Chatellier, Pierre François Plouin, Pierre Corvol, Vicente E. Torres, William F. Young, Kenneth P. Offord, Robert R. Hattery

Research output: Contribution to journal › Letter › peer-review

4 Scopus citations

Abstract

To the Editor: In their intriguing report relating hypokalemia to the development of renal cysts, Torres et al. (Feb. 8 issue)¹ note with surprise a positive correlation of plasma renin activity with the extent of cystic involvement and demonstrate that higher plasma renin activity is the only factor other than a low plasma potassium concentration that is independently predictive of the presence of cysts in a multiple logistic regression analysis. The investigators suggest that uncommon sources, such as renal tubular epithelial cells or the adrenal gland, could contribute to renin levels, but it seems more likely that the renin is.

Original language	English (US)
Pages (from-to)	129-131
Number of pages	3
Journal	New England Journal of Medicine
Volume	323
Issue number	2
DOIs	https://doi.org/10.1056/NEJM199007123230212
State	Published - Jul 12 1990

ASJC Scopus subject areas

Medicine(all)

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title = "Hypokalemia, aldosteronism, and renal cysts",

abstract = "To the Editor: In their intriguing report relating hypokalemia to the development of renal cysts, Torres et al. (Feb. 8 issue)1 note with surprise a positive correlation of plasma renin activity with the extent of cystic involvement and demonstrate that higher plasma renin activity is the only factor other than a low plasma potassium concentration that is independently predictive of the presence of cysts in a multiple logistic regression analysis. The investigators suggest that uncommon sources, such as renal tubular epithelial cells or the adrenal gland, could contribute to renin levels, but it seems more likely that the renin is.",

author = "Weder, {A. B.} and Stewart, {P. M.} and Edwards, {C. R.W.} and Michel Azizi and Christiane Battaglia and Gilles Chatellier and Plouin, {Pierre Fran{\c c}ois} and Pierre Corvol and Torres, {Vicente E.} and Young, {William F.} and Offord, {Kenneth P.} and Hattery, {Robert R.}",

year = "1990",

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doi = "10.1056/NEJM199007123230212",

language = "English (US)",

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TY - JOUR

T1 - Hypokalemia, aldosteronism, and renal cysts

AU - Weder, A. B.

AU - Stewart, P. M.

AU - Edwards, C. R.W.

AU - Azizi, Michel

AU - Battaglia, Christiane

AU - Chatellier, Gilles

AU - Plouin, Pierre François

AU - Corvol, Pierre

AU - Torres, Vicente E.

AU - Young, William F.

AU - Offord, Kenneth P.

AU - Hattery, Robert R.

PY - 1990/7/12

Y1 - 1990/7/12

N2 - To the Editor: In their intriguing report relating hypokalemia to the development of renal cysts, Torres et al. (Feb. 8 issue)1 note with surprise a positive correlation of plasma renin activity with the extent of cystic involvement and demonstrate that higher plasma renin activity is the only factor other than a low plasma potassium concentration that is independently predictive of the presence of cysts in a multiple logistic regression analysis. The investigators suggest that uncommon sources, such as renal tubular epithelial cells or the adrenal gland, could contribute to renin levels, but it seems more likely that the renin is.

AB - To the Editor: In their intriguing report relating hypokalemia to the development of renal cysts, Torres et al. (Feb. 8 issue)1 note with surprise a positive correlation of plasma renin activity with the extent of cystic involvement and demonstrate that higher plasma renin activity is the only factor other than a low plasma potassium concentration that is independently predictive of the presence of cysts in a multiple logistic regression analysis. The investigators suggest that uncommon sources, such as renal tubular epithelial cells or the adrenal gland, could contribute to renin levels, but it seems more likely that the renin is.

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M3 - Letter

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JO - New England Journal of Medicine

JF - New England Journal of Medicine

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Hypokalemia, aldosteronism, and renal cysts

Abstract

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