Animal models of demyelination

M. Kariuki Njenga, Moses Rodriguez

Research output: Contribution to journalArticle

8 Citations (Scopus)

Abstract

In the past year, animal studies on central nervous system demyelination have focused on therapeutic strategies and the functional characterization of T-cells, cytokines, and adhesion molecules. Significant progress has been realized in the treatment of animals with experimentally induced demyelinating disease using glial cell transplants, autoantibodies, cytokines, growth factors, and by inhibiting adhesion molecules and cytokines. In this review, we summarize the important contributions made in those respective areas.

Original languageEnglish (US)
Pages (from-to)159-164
Number of pages6
JournalCurrent Opinion in Neurology
Volume9
Issue number3
StatePublished - 1996

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Demyelinating Diseases
Animal Models
Cytokines
Cell Adhesion Molecules
Neuroglia
Autoantibodies
Intercellular Signaling Peptides and Proteins
Central Nervous System
T-Lymphocytes
Transplants
Therapeutics

ASJC Scopus subject areas

  • Neuroscience(all)

Cite this

Njenga, M. K., & Rodriguez, M. (1996). Animal models of demyelination. Current Opinion in Neurology, 9(3), 159-164.

Animal models of demyelination. / Njenga, M. Kariuki; Rodriguez, Moses.

In: Current Opinion in Neurology, Vol. 9, No. 3, 1996, p. 159-164.

Research output: Contribution to journalArticle

Njenga, MK & Rodriguez, M 1996, 'Animal models of demyelination', Current Opinion in Neurology, vol. 9, no. 3, pp. 159-164.
Njenga MK, Rodriguez M. Animal models of demyelination. Current Opinion in Neurology. 1996;9(3):159-164.
Njenga, M. Kariuki ; Rodriguez, Moses. / Animal models of demyelination. In: Current Opinion in Neurology. 1996 ; Vol. 9, No. 3. pp. 159-164.
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