Adverse outcomes of renovascular hypertension during pregnancy

Bjorg Thorsteinsdottir; Garvan C. Kane; Michael J. Hogan; William J. Watson; Joseph P. Grande; Vesna D. Garovic

doi:10.1038/ncpneph0310

Adverse outcomes of renovascular hypertension during pregnancy

Bjorg Thorsteinsdottir, Garvan C. Kane, Michael J. Hogan, William J. Watson, Joseph P. Grande, Vesna D. Garovic

Research output: Contribution to journal › Article › peer-review

13 Scopus citations

Abstract

Background: A 26-year-old primigravida, with no history of hypertension, presented at 20 weeks of gestation with severe pre-eclampsia. A pelvic ultrasound revealed intrauterine fetal death, probably caused by placental abruption. The pregnancy was terminated by induction with oxytocin, followed by a vaginal breech delivery. The patient remained hypertensive for 8 weeks after delivery. Investigations: Physical examination, laboratory investigation, renal angiogram and renal-vein renin sampling. Diagnosis: An atrophic right kidney secondary to an occluded right renal artery, probably caused by dissected fibromuscular dysplasia; a contralateral high-grade stenosis secondary to fibromuscular dysplasia. Management: Right nephrectomy and angioplasty of the left renal artery.

Original language	English (US)
Pages (from-to)	651-656
Number of pages	6
Journal	Nature Clinical Practice Nephrology
Volume	2
Issue number	11
DOIs	https://doi.org/10.1038/ncpneph0310
State	Published - Nov 2006

Keywords

Hypertension
Pre-eclampsia
Pregnancy outcomes
Renal artery stenosis
Renovascular hypertension

ASJC Scopus subject areas

Nephrology

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10.1038/ncpneph0310

Cite this

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abstract = "Background: A 26-year-old primigravida, with no history of hypertension, presented at 20 weeks of gestation with severe pre-eclampsia. A pelvic ultrasound revealed intrauterine fetal death, probably caused by placental abruption. The pregnancy was terminated by induction with oxytocin, followed by a vaginal breech delivery. The patient remained hypertensive for 8 weeks after delivery. Investigations: Physical examination, laboratory investigation, renal angiogram and renal-vein renin sampling. Diagnosis: An atrophic right kidney secondary to an occluded right renal artery, probably caused by dissected fibromuscular dysplasia; a contralateral high-grade stenosis secondary to fibromuscular dysplasia. Management: Right nephrectomy and angioplasty of the left renal artery.",

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T1 - Adverse outcomes of renovascular hypertension during pregnancy

AU - Thorsteinsdottir, Bjorg

AU - Kane, Garvan C.

AU - Hogan, Michael J.

AU - Watson, William J.

AU - Grande, Joseph P.

AU - Garovic, Vesna D.

PY - 2006/11

Y1 - 2006/11

N2 - Background: A 26-year-old primigravida, with no history of hypertension, presented at 20 weeks of gestation with severe pre-eclampsia. A pelvic ultrasound revealed intrauterine fetal death, probably caused by placental abruption. The pregnancy was terminated by induction with oxytocin, followed by a vaginal breech delivery. The patient remained hypertensive for 8 weeks after delivery. Investigations: Physical examination, laboratory investigation, renal angiogram and renal-vein renin sampling. Diagnosis: An atrophic right kidney secondary to an occluded right renal artery, probably caused by dissected fibromuscular dysplasia; a contralateral high-grade stenosis secondary to fibromuscular dysplasia. Management: Right nephrectomy and angioplasty of the left renal artery.

AB - Background: A 26-year-old primigravida, with no history of hypertension, presented at 20 weeks of gestation with severe pre-eclampsia. A pelvic ultrasound revealed intrauterine fetal death, probably caused by placental abruption. The pregnancy was terminated by induction with oxytocin, followed by a vaginal breech delivery. The patient remained hypertensive for 8 weeks after delivery. Investigations: Physical examination, laboratory investigation, renal angiogram and renal-vein renin sampling. Diagnosis: An atrophic right kidney secondary to an occluded right renal artery, probably caused by dissected fibromuscular dysplasia; a contralateral high-grade stenosis secondary to fibromuscular dysplasia. Management: Right nephrectomy and angioplasty of the left renal artery.

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KW - Pre-eclampsia

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KW - Renal artery stenosis

KW - Renovascular hypertension

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Adverse outcomes of renovascular hypertension during pregnancy

Abstract

Keywords

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